Medically reviewed by Dr. Tom Biernacki, DPM
Board-certified podiatric surgeon | Balance Foot & Ankle, Howell & Bloomfield Hills, MI
Last reviewed: May 2026

| Tendon | Location | Function | Tear Presentation | Clinical Test | Typical Treatment |
|---|---|---|---|---|---|
| FDL (Flexor Digitorum Longus) | Medial ankle behind PTT; plantar foot to lesser toe tips | Plantarflexes toes 2–5; stabilizes forefoot push-off | Medial ankle pain; weakness of lesser toe flexion; often misdiagnosed as PTT injury | Resisted toe flexion test (toes 2–5); tenderness along medial ankle and plantar foot | Repair if acute; FDL-to-FHL tenodesis for chronic/degenerative tears |
| FHL (Flexor Hallucis Longus) | Posterior ankle behind fibula; plantar foot to hallux | Plantarflexes great toe; key for push-off and ballet | Posterior ankle pain; hallux flexion weakness; triggering hallux (stenosing tenosynovitis) | Resisted hallux plantarflexion; positive Hallux Bowstringing test | Endoscopic FHL release for tenosynovitis; tendon repair or hallux fusion for rupture |
| PTT (Posterior Tibial Tendon) | Medial ankle behind medial malleolus; navicular insertion | Supinates foot; primary arch dynamic support | Adult flatfoot; medial ankle pain; progressive arch collapse; positive “too many toes” sign | Single-leg heel rise test (absent in Stage II+) | Stage-based: orthotics → FDL transfer + osteotomy → triple arthrodesis |
| Treatment | Indication | Technique | Expected Outcome | Recovery |
|---|---|---|---|---|
| Conservative (Boot + PT) | Partial tear; low-demand patient; tenosynovitis | 4–6 weeks immobilization; physical therapy for strengthening | 60–70% improvement for partial tears; complete tears rarely heal conservatively | 6–8 weeks to return to activity |
| Primary FDL Repair | Acute complete tear (<6 weeks); adequate tissue quality | End-to-end repair with non-absorbable suture; augmented with Krackow technique | 80–90% good functional outcomes | NWB 6 weeks; 4–6 months to full activity |
| FDL-to-FHL Tenodesis | Chronic FDL tear; degenerated tissue; insufficient stump length | FDL sutured side-to-side to FHL at plantar arch level; FHL drives both | 85% good-to-excellent; minor hallux flexion strength reduction (well-tolerated) | NWB 4–6 weeks; 3–4 months return to sport |
| FDL Reconstruction with Allograft | Large gap; failed prior repair; revision | Tendon allograft bridging defect; secured proximally and distally | 75–80% functional restoration | NWB 8 weeks; 5–6 months to sport |
Quick answer: Flexor Digitorum Longus Tendon Tear Medial Ankle Michigan Podiatrist is a common foot/ankle topic that affects many patients. The 2026 evidence-based approach combines proper diagnosis, conservative-first treatment, and escalation only when needed. We treat this regularly at our Howell and Bloomfield Hills practices. Call (810) 206-1402.
Medically Reviewed | Dr. Tom Biernacki, DPM | Board-Certified Podiatric Surgeon | Balance Foot & Ankle, Michigan

Watch: Heel Bursitis & Achilles Tendon Bursitis [Best HOME Treatment!] — MichiganFootDoctors YouTube
The most important clinical decision with Flexor Digitorum Longus Tendon Tear Medial Ankle Michigan Podiatrist isn’t which treatment to start with — it’s identifying the correct subtype. That changes everything. Call (810) 206-1402.
The most important clinical decision with Flexor Digitorum Longus Tendon Tear Medial Ankle Michigan Podiatrist isn’t which treatment to start with — it’s identifying the correct subtype. That changes everything. Call (810) 206-1402.
FDL Tendon Anatomy and Function
The flexor digitorum longus (FDL) originates on the posterior tibia, passes behind the medial malleolus in the second compartment of the flexor retinaculum (between the posterior tibial tendon anteriorly and the FHL posteriorly), crosses the knot of Henry beneath the navicular, and splits into four slips to the distal phalanges of the lesser toes. Its primary actions are plantar flexion of toes 2–5 and supplemental arch support during push-off. When the FDL is compromised, patients lose lesser toe flexion strength and experience medial arch fatigue.
Causes of FDL Tendon Injury
FDL injuries occur through several mechanisms. Acute tears result from direct trauma, forced dorsiflexion, or avulsion at the knot of Henry. Chronic tendinopathy develops from repetitive overload — common in runners, dancers, and patients with progressive adult flatfoot deformity who overload the medial arch stabilizers. Constriction within the flexor retinaculum can cause a stenosing tenosynovitis similar to trigger finger (trigger toe). FDL pathology frequently coexists with PTTD — both tendons are stressed by progressive flatfoot collapse — and isolated FDL tears are often found incidentally on MRI ordered for suspected PTTD.
Symptoms and How It Differs from PTTD
FDL tendinopathy produces posteromedial ankle pain similar to PTTD but with distinctive features: tenderness is slightly more posterior and inferior; pain is reproduced by resisted lesser toe plantarflexion; and patients may report the toes “not gripping” properly during push-off. Unlike PTTD, single-leg heel-rise is typically preserved unless concurrent flatfoot is severe. A palpable nodule or triggering phenomenon along the tendon course suggests stenosis or longitudinal tear. Differentiation from tarsal tunnel syndrome requires careful neurological examination and EMG/nerve conduction studies.
Diagnosis with MRI and Ultrasound
MRI is the gold standard for FDL pathology assessment — providing high-resolution visualization of tendon continuity, peritendinous fluid, and signal changes indicating degeneration or partial/complete tear. Dr. Biernacki also uses diagnostic ultrasound for dynamic assessment — evaluating tendon gliding, detecting nodules, and guiding injections. Ultrasound is particularly useful for evaluating constriction at the flexor retinaculum and the knot of Henry where the FDL crosses the FHL.
Treatment: Conservative and Surgical Options
Conservative management includes CAM boot immobilization for 4–6 weeks during acute flares; custom orthotics with medial arch support and heel lift to reduce FDL tensile load; physical therapy targeting intrinsic foot strengthening and proximal lower-extremity mechanics; and corticosteroid or PRP injection for persistent tendinopathy. Surgical options for refractory cases include FDL tenosynovectomy and debridement for stenosis; primary repair or imbrication for partial longitudinal tears; and FDL-to-FHL tenodesis for complete or irreparable FDL tears — transferring FDL function to the more powerful adjacent FHL tendon with minimal functional sacrifice, since both tendons share some function through the master knot of Henry.
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✅ Pros / Benefits
- FDL-to-FHL tenodesis provides excellent restoration of lesser toe function with minimal donor site morbidity.
- Dynamic ultrasound assessment detects FDL nodules and stenosis not visible on static MRI sequences.
- Early conservative care with medial arch orthotic support prevents progression from tendinopathy to partial or complete tear.
❌ Cons / Risks
- FDL tears are frequently missed on initial MRI reads by radiologists unfamiliar with forefoot tendon anatomy — clinical correlation and subspecialty review are essential.
- FDL tenodesis to FHL results in some reduction in independent lesser toe flexion strength — typically well-tolerated but relevant for elite athletes.
- Concurrent PTTD (present in many FDL tear patients) requires simultaneous flatfoot reconstruction for optimal outcomes.
Dr. Tom Biernacki’s Recommendation
FDL tears are the hidden pathology I find most often on MRI ordered for ‘PTTD’ — probably 20–30% of what looks like posterior tibial tendon dysfunction on clinical exam has FDL involvement when we image it properly. Missing this diagnosis means incomplete surgical planning and potentially poor outcomes when you reconstruct the flatfoot without repairing the FDL. Accurate imaging interpretation changes the treatment plan.
— Dr. Tom Biernacki, DPM | Board-Certified Podiatric Surgeon | Balance Foot & Ankle
Frequently Asked Questions
How is FDL tendon injury different from posterior tibial tendon dysfunction?
Both cause medial ankle pain, but FDL injuries involve the tendon slightly more posterior and affect lesser toe flexion rather than single-leg heel-rise ability. MRI differentiates the two — PTTD shows posterior tibial tendon pathology; FDL tears show signal changes in the adjacent second compartment tendon. Both often coexist and require combined surgical planning.
Can I walk on a partially torn FDL tendon?
Yes — partial FDL tears typically allow continued weight-bearing with pain modification. A CAM boot or custom orthotic reduces load on the tendon during healing. Complete tears may cause more significant functional deficits but are still usually ambulatory. Dr. Biernacki will guide activity modification based on tear severity on MRI.
How long is recovery after FDL repair surgery?
FDL tenosynovectomy without repair: 4–6 weeks non-weight-bearing then progressive return to activity by 3–4 months. FDL repair or tenodesis: 6–8 weeks non-weight-bearing, PT beginning at 8 weeks, return to full activity by 5–6 months. Concurrent flatfoot reconstruction adds complexity and extends recovery.
Is the FDL the same as the FHL?
No — the FDL (flexor digitorum longus) flexes toes 2–5, while the FHL (flexor hallucis longus) flexes the big toe. They cross each other at the ‘knot of Henry’ beneath the navicular. FHL tears are more common in ballet dancers; FDL tears more common in runners and flatfoot patients. Both run behind the medial malleolus in adjacent retinacular compartments.
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When should I see a podiatrist?
If symptoms persist past 2 weeks, affect your normal activity, or are accompanied by red-flag symptoms (warmth, redness, swelling, inability to bear weight).
What does treatment cost?
Most diagnostic visits and conservative treatments are covered by Medicare and major insurers. Out-of-pocket costs vary by your specific plan.
How quickly can I get an appointment?
Most non-urgent cases see us within 5 business days. Urgent cases (sudden pain, possible fracture) typically same or next business day.
In-Office Treatment at Balance Foot & Ankle
If home treatment isn’t providing relief for your Achilles tendon conditions, our podiatry team at Balance Foot & Ankle can help with same-day evaluations and advanced in-office care.
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Dr. Tom Biernacki, DPM is a board-certified foot & ankle surgeon (ABFAS & ABPM) at Balance Foot & Ankle Specialists in Southeast Michigan. With over a decade of clinical experience, he specializes in heel pain, bunions, diabetic foot care, sports injuries, and minimally invasive surgery. Dr. Biernacki is a member of the APMA and ACFAS, and his patient education content on MichiganFootDoctors.com and YouTube has made him one of the most-followed foot & ankle educators on YouTube.